2007: Systemic Sclerosis, Fibrosing Syndromes and Raynaud's: Pathogenesis, Animal Models and Genetics
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Systemic Sclerosis, Fibrosing Syndromes and Raynaud's: Pathogenesis, Animal Models and Genetics
33
Association between Genetic Polymorphisms in the Surfactant Protein Genes and Pulmonary Fibrosis in Systemic Sclerosis
34
The Shared Amino Acid Sequence “FLED” of Some DRB1 and DRB5 Alleles is Associated with Systemic Sclerosis
35
Whole Genome Association by DNA Pooling Leads to Interesting Candidate Genes for Systemic Sclerosis
36
Gene-Gene Interactions Mediating Susceptibility to Scleroderma: Investigation of A 3 Gene Network
37
Comparative Analysis of Dermal Fibroblast Gene Expression Profiles from Patients with Systemic Sclerosis and Hypertrophic Burn Scars
38
Systemic Sclerosis and Systemic Lupus Erythematosus have Distinct Gene Expression Profiles in the Peripheral Blood Cells
40
Molecular Subtypes of Systemic Sclerosis (SSc) From Gene Expression Signatures
41
Adenosine A
2A
Receptor Occupancy Regulates Expression of Multiple Genes Involved in Fibrosis
42
Pharmacological Blockade of Adenosine A
2A
Receptors Reduces IL-13 Production and Fibrotic Features in a Novel Model of Skin Fibrosis
43
Nilotinib and Dasatinib, Two Novel and More Potent Inhibitors of C-abl and Pdgf Signalling, for the Treatment of Experimental Dermal Fibrosis
44
The Antifibrogenic Effects of Trichostatin A are Mediated Through the Down-Regulation of Histone Deacetylase 7
45
Altered in vivo Dermal and Epidermal TGFβ Signalling in TβRII Conditional Knockout Mice
46
Role of Type I Interferons in a Murine Model of Scleroderma
47
Development of Smad7 Conditional-Knockout Mice and Its Altered Fibrotic Responses in Fibroblast in Vitro
48
Intercellular Adhesion Molecule-1 Deficiency Attenuates the Development of Skin Fibrosis in Tight-skin Mice
49
Chemokine Receptors CCR2 and CX3CR1 Regulate Skin Fibrosis in a Cytokine-induced Scleroderma Mouse Model
50
Baff Antagonist Attenuates the Development of Skin Fibrosis in Tight-Skin Mice
51
An
in vitro
Study of the Effect of Gadolinium Compounds on Normal Human Peripheral Blood Mononuclear Cells.
52
Macrophage Migration Inhibitory Factor (MIF) Production by T Cells is Strongly Upregulated by Allograft Inflammatory Factor-1 (AIF-1) Expression and Contributes to the Profibrotic Effects of AIF-1
53
IGFBP-5 is a Novel Chemoattractant for Immune Cells and Fibroblasts and Plays a Role in the Inflammatory and Fibrotic Phases of Pulmonary Fibrosis
54
IGFBP-5 Induces Extracellular Matrix Production via Egr-1 and MAPK-Dependent Pathway
55
Insulin-Like Growth Factor-ii (IGF-II) is Aberrantly Expressed in Scleroderma-Associated Pulmonary Fibrosis and Contributes tt Abnormal Matrix Production
56
Assembly of Fibrillin-1 and Elastic Fibers is Repressed by CCN3, a Growth Factor Upregulated in Tsk Mice and Downregulated in a Marfan Syndrome Model: Novel Molecular Insights into Extracellular Matrix Remodeling in Connective Tissue Disorders
57
Heparan Sulphate-Dependent ERK Activation Contributes to the Over-Expression of Fibrotic Proteins and the Enhanced Contraction by Lesional Dermal Scleroderma Fibroblasts of Their Extracellular Matrix
58
Up-take of Silica and Carbon Nanotubes by Human Macrophages Induces Activation of T Cells and Fibroblasts i
n vitro -
Potential Implication for
Pathogenesis of Fibrosing Diseases
59
Differential Dynamic Properties of the TGFB Pathway between Normal Fibroblasts and Scleroderma Fibroblasts in Response to Perturbation by Environmental Stimuli
60
DNA Methylation Induction by a Circulating Activity in SSc Sera: Possible Role for Oxidation and Autoantibodies Related Mechanisms
61
High Levels of Circulating Endothelial Progenitor Cells and Functional Properties of Their Late Outgrowth in Systemic Sclerosis
62
Disturbed Sera Angiogenic Markers in Systemic Sclerosis
63
Reactive Oxygen Species Production in Scleroderma
64
Paradoxical Increase in Monocytic Progenitors for Endothelial Cells in Patients with Systemic Sclerosis
65
Anti-Fibroblast Antibodies (AFA) Detected by Cell-Based Elisa in Systemic Sclerosis (SSc) Sera Preferentially Induce Pro-Fibrotic Chemokine Production in Human Dermal Fibroblasts
66
Serum COMP in Systemic Sclerosis - A Promising New Biomarker for Skin Involvement
67
Involvement of IC-IL-1RA in the Transition of Low Density Seeded Fibroblasts into Myofibroblasts
68
Collagenase Expression is Inhibited in Fibroblasts from Fibrotic, but Not Non-fibrotic, Scleroderma Skin: The Role of Intracellular Interleukin 1 Receptor Antagonist
69
Anti-Topoisomerase I, but not Anti-Centromere, Autoantibodies Induce IFN-a through a Toll Like Receptor Pathway: Correlations with Disease Manifestations
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