2008: RA: Human Etiology and Pathogenesis - Molecular and Cellular Mechanisms
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RA: Human Etiology and Pathogenesis - Molecular and Cellular Mechanisms

Monday, October 27, 2008
1250
Ectopic Lymphoid Structures Expressing AID Drive Self Sustained in situ Production of Autoantibodies in Rheumatoid Synovium
1251
Functional Implications Of Defective Telomerase Activity In Rheumatoid Arthritis - Impaired Survival Of Naïve T Cells
1252
Potent Induction Of Th17 Cells By Synovial Monocytes From Active Rheumatoid Arthritis Patients
1253
Increased Th17 Cell Frequency and Effector Functions in Patients with Early Rheumatoid and Psoriatic Arthritis
1254
Dopamine Functions As A Dendritic Cells-mediated Th2-polarizing Factor And Plays A Role For The Pathogenesis Of Rheumatoid Arthritis
1255
Mechanism Of Increased Metalloproteinase And JNK Activation In Rheumatoid Arthritis (RA) Synoviocytes
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